Buck Research Focuses On Risk Factor For Parkinson’s Disease
A new study demonstrates that high levels of MAO-B, an enzyme that regulates nerve activity in the brain, cause Parkinson’s-like symptoms in mice genetically engineered to overexpress the protein. Furthermore, drugs currently used as an adjunct therapy for Parkinson’s in humans prevented the development of Parkinson’s symptoms in these same animals.
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Filed under: Parkinson's Disease on February 26th, 2008